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Regulation of cyclin expression & effects on root apical growth

Regulation of cyclin expression & effects on root apical growth

Thomas Potuschak (ORCID: )
  • Grant DOI 10.55776/J1907
  • Funding program Erwin Schrödinger
  • Status ended
  • Start April 8, 2000
  • End April 8, 2001
  • Funding amount € 26,162
  • Project website

Disciplines

Biology (100%)

Keywords

    PANT DEVELOPMENTAL BIOLOGY, PLANT GENETICS, MOLECULAR BIOLOGY

Abstract

Erwin Schrödinger Fellowship J 1907 Regulation of cyclin expression & effects on root growth Thomas POTUSCHAK 06.03.2000 Ectopic expression of the Arabidopsis cyclin B1;1 accelerates root growth without altering the pattern of lateral root formation (Doerner et all, 1996). Thus cyclin expression is a limiting factor for root apical growth and cyclin expression might be a target for growth control pathways in plants. Functional analysis with promoter deletion variants placed upstream of either the same mitotic cyclin or an reporter gene in both transgenic cell cultures as well as transformed plants has revealed the presence of both quantitative and qualitative promoter elements. The qualitative promoter elements are responsible for stimulating cyclin expression at the proper time in the cell cycle, around the G2/M transition, as well as they are sufficient to direct expression to the meristems. However, the magnitude of cyclin steady state RNA accumulation is approximately tenfold less than that observed with the full-length promoter. Further analysis has revealed the presence of positive and negative regulatory elements, which serve to modulate the amplitude of cyclin transcription. The cognate DNA binding comlexes appear to be the best candidates for effectors of growth control pathways. I want to characterise the previously identified promoter elements in greater detail and identify the growth control pathways that act on them to affect root apicat meristem activity via regulation of cyclin transcription. In order to dissect these pathways it is of special interest to me to clone factors that bind to regulatory factors within the cyclin promoter. Cloning of these factors will allow the identification of the pathways that act through them and will enable us to study directly how they effect the regulation of cyclin transcription.

Research institution(s)
  • Universität Wien - 10%
  • University of Edinburgh - 100%

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